Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. Alternatively, our ability to rescue SERT function in KI midbrain synaptoneurosomes by the inhibition of FAK implies elevated FAK signaling downstream of your Pro32Pro33 mutant, as verified by enhanced pFAK localization in 5-HT synapses. Our information https://tims653tep4.plpwiki.com/user